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Privacy Policy - Royal Queen Seeds

To ensure a safe online environment and guarantee adequate data protection, we strictly comply with all legal requirements. In this privacy statement, we provide information about how and for what purpose data is collected, safety measures, storage periods and contact details.


COMPANY NAME: SNORKEL SPAIN, SL (hereinafter ROYAL QUEEN SEEDS)

C/ Vilar d'Abdelà, 5 (nave 1) CP: 08170 de Montornès del Vallès

+34 937 379 846

[email protected]


The present Privacy Policy sets out the terms on which we will treat personal data at ROYAL QUEEN SEEDS; this includes any personal data collected through our website https://www.royalqueenseeds.com/ as well as any other data we process in the course of our business activities.

ROYAL QUEEN SEEDS collects the following personal data for the purposes listed below:

SECTION 1 – PERSONAL INFORMATION WE COLLECT

1.1 Account purchases

Account purchases can only be made if you are in possession of a personal account. When you create an account or purchase something from our shop, as part of the buying and selling process we collect the following personal information that you provide to us:

  • First and last name
  • Home and billing address
  • Telephone number
  • Gender
  • IP address
  • Email address
  • Date of birth

This information is required for delivery. In addition, when you browse our shop, we automatically receive the Internet Protocol (IP) address of your computer. Based on this information, we can optimise your online experience and at the same time protect our online environment.

Purpose of data collection

We collect and store account-related data for the following purposes:

(a) to carry out obligations arising from any contracts between you and us, and to provide you with information, products and services that you may request from us;

(b) to set up, manage and communicate with you about your account and your orders;

(c) to conduct market research and analysis;

(d) to confirm your age and identity, and to detect and prevent fraud.

1.2 Newsletters

With your explicit permission, we may send you newsletters about our shop, new products and other updates. We send newsletters based on your explicit consent. In the event that you purchase a product, and in accordance with current regulations, we may send you commercial communications in accordance with the legitimate interest of our company, always about products or services similar to those you have purchased or contracted. In any case, you may exercise your right of opposition through the channels announced in this Privacy Policy. The following information is collected in relation to the newsletter:

  • First & last name
  • Gender

We do not need to know the sex of the person in order to send the newsletter (data minimisation: by law we must ask for data that is strictly necessary to provide the service, and in this case knowing the sex is not necessary to send the newsletter).

  • Email address

Purpose of data collection

The data collected is used to:

(a) personalise our emails, including your name and gender;

(b) provide gender-specific content.

You can withdraw your consent at any time by using the link provided in the newsletter or the contact information provided in section 2.

1.3 Customer service and contact form

In order to provide appropriate support, our customer service employees have access to information related to the account. Consequently, their support will be highly effective and friendly. The data provided in our contact form is used by our CRM provider, SuperOffice. We will only use your details to respond to your message.

SECTION 2 – LEGITIMATE INTEREST

If you have purchased any of our products, please note that we may process your personal data for promotional purposes, based on Royal Queen Seeds' legitimate interest only to offer you products or services from our company and about products or services similar to those you have purchased. You may exercise your right to opt out of future messages by the means set out in this Privacy Policy or through any notification you receive.

2.1 How do you withdraw consent?

If you change your mind, you can withdraw your consent for us to contact you for the purpose of collecting, using, or disclosing your data at any time by reaching out to us at: [email protected].

SECTION 3 – DISCLOSURE

We may disclose your personal information if we are required to do so by law or if you breach our Terms of Service.

SECTION 4 – HOW LONG DO WE KEEP YOUR DATA?

At Royal Queen Seeds, we will not retain your data for longer than is necessary for the purposes described in this Policy. Different retention periods apply for different types of data; however, the longest period we will normally hold any personal data is 10 years.

4.1 Account information

Data relating to the account remains relevant for as long as the consumer is in possession of an account. Therefore, the data remains documented for as long as the account exists. When our customers delete an account, the associated data will be deleted within a reasonable period of time. Requests regarding the inspection or correction of stored personal data or the deletion of an account can be sent to [email protected].

4.2 Newsletters

In the event that you give us your consent to inform you about our products or services, we will keep your data until you express your wish not to receive any further communications from us. However, we regularly (every month) carry out a relevance check. Registered customers (and their personal information) will be deleted whenever customers do not reply to our request. In addition, our newsletter mailing has an opt-out feature. Consumers can withdraw their consent by using this opt-out feature.

SECTION 5 – COOKIES

Cookies are small information files that notify your computer of previous interactions with our website. These cookies are stored on your hard drive, not on our website. Essentially, when you use our website, your computer displays its cookies to us, informing our site that you have visited before. This allows our website to function more quickly and remember aspects related to your previous visits (such as your username), making your experience more convenient. At Royal Queen Seeds, we use two types of cookies: functional and analytical.

5.1 Functional Cookies

Functional cookies are used to enhance your online experience. Among other things, these cookies track what is added to your shopping cart. The use of these cookies does not require prior authorization.

5.2 Analytical Cookies

Analytical cookies are used for research and market analysis. The data collected with these analytical cookies is anonymous, making it unusable for third parties. The use of these cookies does not require prior authorization.

SECTION 5 – THIRD-PARTY SERVICES

Third-party services are required to conduct transactions and provide our services. In general, the third-party providers we use will only collect, use and disclose your information to the extent necessary to enable them to perform the services they provide to us.

However, certain third-party service providers, such as payment gateways and other payment transaction processors, have their own privacy policies regarding the information we must provide to them for your transactions.

We encourage you to read the privacy policies of these providers so that you can understand how these providers will handle your personal information.

In particular, certain suppliers may be located or have facilities located in a different jurisdiction than yours or ours. Therefore, if you choose to proceed with a transaction involving the services of a third party, your information may be subject to the laws of the jurisdiction in which that service provider or its facilities are located.

Once you leave our website or are redirected to a third-party website or application, you are no longer governed by this Privacy Policy or the Terms of Service of our website.

Web analytics service (anonymous data)

On this website we have integrated an element of a web analytics service (with anonymisation functionality). Web analytics can be defined as the gathering, processing and analysis of data about the behaviour of visitors to websites. An analytics service collects, among other things, data about which website a person came from (the so-called referrer), which sub-pages they visited or how often and for how long they visited a sub-page. Web analytics is mainly used for website optimisation and for a cost–benefit analysis of internet advertising.

Courier service

To complete deliveries we use a courier service. This courier service carries out the delivery between our company and the consumer's home. To complete these logistics, the company requires access to the consumer's name and address information.

Mailing service

Royal Queen Seeds uses a third-party mail service provider to send its newsletter. This provider has access to limited account information related to opt-in consent (e.g. email address).

Marketing services

Royal Queen Seeds has the support of a company that specialises in marketing and communication activities. Their access to personal information is very limited and mostly anonymous.

Payment services

At Royal Queen Seeds we use external payment services to handle our transactions (e.g. credit card payments).

SECTION 6 – SECURITY

To protect your personal information, we take reasonable precautions and follow industry standard best practices to ensure that it is not inappropriately lost, misused, accessed, disclosed, altered or destroyed.

If you provide us with your credit card information, the information is encrypted using secure socket layer technology (SSL) and stored using AES-256 encryption. Although no method of transmission over the internet or electronic storage is 100% secure, we follow all PCI-DSS requirements and implement additional industry standards that are commonly accepted. Information related to the account is protected with a hashing method. This method transforms the information into a generated hash. As a result, confidential information is protected and invisible, even to us. In addition, our databases are exceptionally protected against unauthorised access. For example, access to the database is only possible and permitted via approved IP addresses (e.g. from Royal Queen Seeds headquarters). Other attempts and addresses are rejected at all times.

Furthermore, data is anonymised as much as possible, so it cannot be directly linked to a specific consumer. With this data, however, we may be able to carry out market research and analysis. In addition, the third parties concerned (e.g. mailing service) are examined prior to our collaboration, comply with the GDPR from the EU and receive a processing agreement. Within Royal Queen Seeds, employees are assigned different access permissions. Specific permission provides access only to information that is strictly required to perform a task. Digital security measures are subject to change and must meet high requirements to ensure the safety of online customers. That is why, at Royal Queen Seeds, we appoint a security officer. Regular verification and improvement of security measures (where necessary) are part of the role.

SECTION 7 – CHANGES TO THIS PRIVACY POLICY

We reserve the right to modify this Privacy Policy at any time, so please check back regularly. Changes and clarifications will be effective immediately upon being published on the Website. If we make substantial changes to this policy, we will notify you here that it has been updated so you will know what information we collect, how we use it and under what circumstances, if any, we use and/or disclose it.

SECTION 8 – YOU HAVE THE RIGHT TO:

  • Revoke the consent you have granted;
  • Access personal data;
  • Rectify personal data;
  • Delete personal data;
  • File a complaint or a writ for the protection of your rights before the Spanish Data Protection Agency;
  • Be notified of any security incident that may affect your rights;
  • Limitation of processing;
  • Portability.

What are Cannabinoids and why are they important?

What are Cannabinoids and why are they important?

Contents:

All cannabis enthusiasts will have heard of THC, known to science as (–)-trans-Δ⁹-tetrahydrocannabinol. This molecule is responsible for the distinguished psychoactive effects felt after smoking a spliff or puffing on a vaporizer. Many will also have heard of CBD, or cannabidiol, by this point.

Both THC and CBD belong to a chemical class called cannabinoids. This class is composed of a diverse array of molecules that act upon cannabinoid receptors. THC and CBD are phytocannabinoids, meaning they are synthesised in and derived from the cannabis plant. Over 100 of these molecules have been identified to date. Another cannabinoid type are endocannabinoids, which are created in the body, and include anandamide and 2-AG. Finally, there are those cannabinoids that are synthesised in laboratories; these are called synthetic cannabinoids.

In this article, our focus is on the phytocannabinoids. Many of these molecules have been studied by science, and have displayed an impressive therapeutic potential to boot. Before we delve into individual cannabinoids, let’s look at the function of cannabinoids and how they are created.

WHAT ARE THE FUNCTIONS OF CANNABINOIDS?

The answer to this query all depends on the life form in question. Plants produce cannabinoids as secondary metabolites; they aren’t directly involved in the growth or development of the plant, and are believed to instead play a protective role against pests, diseases, and ultraviolet radiation. For example, the phytocannabinoid THCA has been shown to prompt the death of insect cells, suggesting it serves as a defence mechanism against certain species that decide to make lunch out of cannabis flowers and leaves.

In humans, cannabinoids work in a very unique and specific manner. Some of them directly interact with the endocannabinoid system, an internal network of receptor sites found on many cell types. These receptors, namely the CB1 and CB2 receptors, are found throughout the central nervous system and immune system. Certain cannabinoids, for example THC, produce psychoactive and therapeutic effects by interacting with these sites.

The endocannabinoid system plays a regulatory role within the body and helps the organism maintain a state of homeostasis—the biological dynamic equilibrium our bodies require to function optimally. The endocannabinoid system has been found to play a role in the regulation of the endocrine system and metabolism.

The aforementioned endocannabinoids anandamide and 2-AG serve as signalling molecules that interact with these receptors to allow the endocannabinoid system to exert these functions[1]. The molecular similarity between phytocannabinoids and endocannabinoids is what makes it possible for these external counterparts to induce physiological effects.

Psychoactive Cannabinoids

Non-Psychoactive Cannabinoids

HOW CANNABINOIDS ARE CREATED: BIOSYNTHESIS

It’s both important and interesting to know how cannabinoids are created. Have you ever looked closely at a cannabis flower and witnessed what appears to be thousands of glistening jewels? These translucent, mushroom-shaped structures are called glandular trichomes, and are responsible for producing a resin rich in cannabinoids, terpenes, and more.

Cannabis plants direct nutrients to these glands, specifically the secretory cells, where they are turned into the precursors for cannabinoid production. These precursors are then transported to the secretory vesicles, where they are built into trichomes and eventually excreted.

All cannabinoids start off life as CBGA (cannabigerolic acid). This cannabinoid acid is formed via the combination of cannabinoid precursors olivetolic acid and geranyl pyrophosphate after an array of chemical processes[2]. CBGA is the precursor of cannabinoid biosynthesis, and is used as a substrate to create other cannabinoids via enzymes CBDAS, CBCAS, and THCAS. Depending on the enzyme used, the outcome of this reaction is either CBDA (cannabidiolic acid), CBCA (cannabichromenic acid), or THCA (tetrahydrocannabinolic acid).

CANNABINOID ACIDS VS. CANNABINOIDS

Cannabinoids occur naturally within cannabis plants in their acid form. Their molecular structure is different because of the addition of a carboxyl group. However, this group has some volatility and can be removed via heat or degradation. For example, raw cannabis flowers contain THCA as opposed to THC. When these flowers are placed into a joint and a flame is applied, the heat catalyses the release of a carboxyl group, and inactive THCA becomes psychoactive THC. This process is known as decarboxylation.

INTRODUCING THE MAJOR CANNABINOIDS

Of the 100+ discovered cannabinoids, only a few have been studied to a relatively in-depth degree. Here, we’ll take a look at the characteristics of each, and see what the research says about their therapeutic potential. We’ll start with the most familiar, and then delve into some of the lesser-known molecules.

THC

THC Therapeutic Effects

THC is perhaps the most famous of all cannabinoids, as well as the most controversial. This chemical is primarily responsible for the mind-altering high induced when smoking or ingesting cannabis, and also has a variety of therapeutic uses. THC is the most abundant cannabinoid in chemotypes (a descriptive term used to signify the chemical makeup of a cultivar in regards to its secondary metabolites) selectively bred for inducing a psychoactive effect. It is THC's interaction with the CB1 receptor within the central nervous system that gives rise to its psychotropic action.

One of the most staggering discoveries regarding THC is its ability to induce apoptosis (controlled cell death) in tumour cells, whilst protecting healthy cells from cell death. This research[3] has been performed in animal models, but more research is desperately needed in order to explore the therapeutic value of THC in this domain.

THC interacts with both the CB1 and CB2 receptors. Through this mechanism, the molecule has also been shown[4] to modulate pain, spasticity, sedation, appetite, and mood. For example, research[5] published in the journal _Clinical Therapeutics_ found a significant reduction in pain in patients with progressive multiple sclerosis immediately following the administration of an oral preparation of THC.

Additionally, THC also displays neuroprotective effects, and has even been shown[6] to lower amyloid beta levels in vitro (outside of a living organism), suggesting it could play a role as a potential therapeutic in Alzheimer’s disease.

Finally, and quite astonishingly, THC is a potent anti-inflammatory that has shown[7] 20 times the anti-inflammatory effects of aspirin.

CBD

CBD Therapeutic Effects

CBD is the second most abundant cannabinoid in many selectively bred cannabis chemotypes, and the main cannabinoid constituent in cultivars bred for medical purposes. CBD has gained massive traction in recent years because of its non-intoxicating nature, good safety profile, and impressive medicinal value. Many regions that prohibit cannabis have designed legislation to allow the sale of CBD products—as long as THC levels are below a certain threshold dictated by local law.

One of the most interesting studies thus far regarding the cannabinoid involves tumours. A study[8] published in Molecular Cancer Therapeutics examined the impact of CBD on a breast cancer cell line. The results pointed to a complex mechanism, highlighting “the value of continued investigation into the potential of CBD”.

CBD does interact with the endocannabinoid system, yet has a low affinity for both the CB1 and CB2 receptors. The cannabinoid largely exerts its effects by binding to a range of other receptors, including serotonin and vallinoid receptors.

However, the molecule is a CB1 antagonist[9], meaning it blocks other molecules, like THC and 2-AG, from binding to these receptor sites. As a negative allosteric modulator[10] of CB1, CBD has been shown to reduce some of the adverse psychological effects of THC.

Moreover, CBD is indirectly able to boost levels of endocannabinoids in the body, potentially accounting for its analgesic and antipsychotic[11] effects. Endocannabinoids anandamide and 2-AG are metabolised (broken down) by the enzyme FAAH (fatty acid amide hydrolase). Intriguingly, CBD inhibits FAAH, temporarily boosting anandamide levels within the periaqueductal grey.

CBD is also associated with anticonvulsant effects and an ability to reduce certain types of seizures. Research[12] published within the journal _Neuropharmacology_ investigated the clinical benefits of both CBD-rich cannabis extracts and purified CBD in treatment-resistant epilepsy. Data from 670 patients was analised, and it was found that approximately 60% of patients reported improvement in seizure frequency. Interestingly, it was found that CBD-rich extracts were associated with more improvements than isolated CBD. The researchers state this difference could have been due to the entourage effect, the ability of terpenes and cannabinoids to synergise.

CBD has also displayed[13] anxiolytic, immunosuppressive, neuroprotective, and antioxidant properties in research settings.

CBG

CBG Therapeutic Effects

CBG is found is large quantities in many cannabis cultivars in full bloom, and has been found to possess numerous therapeutic applications. The cannabinoid is a vanilloid and CB1 receptor antagonist and works[14] in a similar way to CBD, namely by inhibiting the reuptake of anandamide. As an antagonist, it works to block other molecules from binding to receptors and doesn’t activate them. In regards to the CB1 receptor, this makes CBG non-mind-altering.

Like its aforementioned counterparts, CBG displays potential in the realm of tumours. A paper[15] published in the journal _Carcinogenesis_ investigated the antineoplastic effects of CBG in colon cancer in mice. The results showed the cannabinoid to promote apoptosis and reduce cellular growth. The researchers concluded that CBG should be considered as a future treatment. In other research, CBG, along with other cannabinoids, displayed[16] inhibition of cell growth in a breast cancer model.

CBG has also been proven[17] to exert painkilling, antidepressant, and antibacterial effects. It’s also been found to have a positive effect on psoriasis by stopping the excessive growth of certain skin cells.

CBN

CBN Therapeutic Effects

CBN isn’t biosynthesised within the trichomes of cannabis plants. Instead, it’s the result of the degradation of THC by way of oxidation. After exposure to prolonged storage or exposure to heat, light, and oxygen, THC is broken down into CBN.

The full effects profile of CBN is yet unclear, but it is reported to induce sedation. Ever smoked a strain that made you feel extra sleepy? This might be due to high levels of specific terpenes, or maybe because the buds in question were stored for a while and exposed to some level of degradation.

Even this cannabinoid, created through oxidation, has demonstrated some rather impressive medicinal qualities. Much like CBG, CBN has displayed some promise for psoriasis. This is because both appear[18] to reduce the overproduction of skin cells called keratinocytes, which contribute towards inflammation. CBN also features anticonvulsant and antibacterial properties[19].

CBC

CBC Therapeutic Effects

CBC levels vary dramatically within cannabis plants. Some samples show minimal levels, while cultivars derived from selective breeding programs produce much higher quantities, leading to the cannabinoid becoming one of the most abundant within cannabis. Interestingly, derivatives of the cannabinoid can be found elsewhere in nature, including Rhododendron species and some fungi.

CBC has been shown to possess antinociceptive properties, meaning it works to block the detection of painful stimulus, a desirable trait in some pain-killing drugs. The cannabinoid also has anti-inflammatory effects and has even been shown to increase the effects of THC in vivo (in living organisms). This effect could be of considerable interest to breeders seeking to produce cultivars with potent psychoactive effects.

CBC may exert these effects[20] because of its ability to bind to the CB2 receptor. CBC is a selective CB2 receptor agonist and may contribute to the therapeutic potential of some cannabis formulas by addressing inflammation via this receptor site.

THCV

THCV Therapeutic Effects

As the name suggests, THCV (tetrahydrocannabivarin) is a molecule similar to THC. The difference is that the molecule is a propyl analogue of THC. THCV interacts[21] with both the CB1 and CB2 receptors. The cannabinoid is a partial agonist of the CB2 receptor, meaning that it has some affinity for the receptor. THCV displays varying behaviour in terms of its relationship to the CB1 receptor. At low doses, it works as an antagonist, blocking some receptor activity. However, at higher doses, the cannabinoid becomes a CB1 receptor agonist and starts to activate it.

This relationship with the CB1 receptor is why THCV's psychoactive properties have come into question. THC achieves its effects at the CB1 receptor even at low doses. THCV is indeed psychoactive, but high doses are required to turn the table from it blocking the receptor to activating it. In contrast, low doses are capable of suppressing appetite and reducing the psychoactive effects of THC.

As far as its therapeutic qualities[22] go, THCV has been found to induce weight loss in obese mice, and to possess anticonvulsant and anti-inflammatory effects.

CBDV

CBDV Therapeutic Effects

CBDV is a propyl analogue of CBD. Like CBD, CBDV has a low affinity for cannabinoid receptors and also inhibits the breakdown of the endocannabinoid anandamide. CBDV also interacts with vanilloid receptors and the debated third endocannabinoid receptor GRP55. Early research[23] has found CBDV to delay neurological defects in mice, but only in a short-term manner after administration. CBDV has also shown anticonvulsant properties[24], and may even outperform CBD is this domain, a cannabinoid famed for this application. CBDV also displays promise for easing nausea and vomiting.

THCA

THCA Therapeutic Effects

THCA is a cannabinoid acid synthesised within the trichomes of cannabis plants. The molecule is converted to THC via heat or long-term degradation. THCA is non-intoxicating and is a weak agonist of both the CB1 and CB2 receptors. Research indicates that it has anti-inflammatory, neuroprotective, antineoplastic, and immunomodulatory effects. A study[25] conducted on obese mice found that the cannabinoid acid is also capable of decreasing fat tissue and preventing metabolic disease.

CBDA

CBDA Therapeutic Effects

CBDA is the cannabinoid acid precursor of CBD prior to decarboxylation. This molecule interacts with serotonin, vanilloid, and GPR55 receptors, similar to its activated counterpart CBD. And much like CBD, numerous studies are underway examining the compound’s influence on well-being. Research[26] published within the journal Toxicology Letters claimed to find an interaction between CBDA and certain types of breast cancer cells.

THE FUTURE OF CANNABINOID RESEARCH

In this article, we have briefly glanced at the most studied cannabinoids, yet the research surrounding them is far from conclusive. More large-scale, double-blind, placebo-controlled clinical studies will help us to determine with more certainty what each individual cannabinoid is capable of, as well as how they all work in tandem. With over 100 phytocannabinoids identified in the cannabis plant, the future certainly holds some fascinating discoveries.

External Resources:
  1. [The role of the endocannabinoid system in the regulation of endocrine function and in the control of energy balance in humans]. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  2. The Biosynthesis of Cannabinoids - ScienceDirect https://www.sciencedirect.com
  3. Cannabis and Cannabinoids (PDQ®)–Health Professional Version - National Cancer Institute https://www.cancer.gov
  4. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3165946/
  5. Effects on Spasticity and Neuropathic Pain of an Oral Formulation of Δ9-tetrahydrocannabinol in Patients With Progressive Multiple Sclerosis - ScienceDirect https://www.sciencedirect.com
  6. The potential therapeutic effects of THC on Alzheimer's disease. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  7. Thieme E-Journals - Planta Medica / Abstract https://www.thieme-connect.de
  8. Peter OBryan http://mct.aacrjournals.org
  9. Cannabidiol displays unexpectedly high potency as an antagonist of CB1 and CB2 receptor agonists in vitro. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  10. Cannabidiol is a negative allosteric modulator of the cannabinoid CB1 receptor. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  11. Cannabidiol enhances anandamide signaling and alleviates psychotic symptoms of schizophrenia | Translational Psychiatry https://www.nature.com
  12. Frontiers | Potential Clinical Benefits of CBD-Rich Cannabis Extracts Over Purified CBD in Treatment-Resistant Epilepsy: Observational Data Meta-analysis | Neurology https://www.frontiersin.org
  13. Cannabis Pharmacology: The Usual Suspects and a Few Promising Leads - ScienceDirect https://www.sciencedirect.com
  14. Cannabis Pharmacology: The Usual Suspects and a Few Promising Leads. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  15. Colon carcinogenesis is inhibited by the TRPM8 antagonist cannabigerol, a Cannabis-derived non-psychotropic cannabinoid | Carcinogenesis | Oxford Academic https://academic.oup.com
  16. Cannabis Pharmacology: The Usual Suspects and a Few Promising Leads. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  17. Cannabis Pharmacology: The Usual Suspects and a Few Promising Leads - ScienceDirect https://www.sciencedirect.com
  18. Cannabinoids inhibit human keratinocyte proliferation through a non-CB1/CB2 mechanism and have a potential therapeutic value in the treatment of psoriasis - ScienceDirect https://www.sciencedirect.com
  19. Cannabis Pharmacology: The Usual Suspects and a Few Promising Leads. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  20. Cannabichromene is a cannabinoid CB2 receptor agonist | bioRxiv https://www.biorxiv.org
  21. Error - Cookies Turned Off https://bpspubs.onlinelibrary.wiley.com
  22. Cannabis Pharmacology: The Usual Suspects and a Few Promising Leads. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  23. SAGE Journals: Your gateway to world-class journal research https://journals.sagepub.com
  24. Cannabis Pharmacology: The Usual Suspects and a Few Promising Leads. - PubMed - NCBI https://www.ncbi.nlm.nih.gov
  25. Tetrahydrocannabinolic Acid a (THCA-A) Reduces Adiposity and Prevents Metabolic Disease Caused by Diet-Induced Obesity | bioRxiv https://www.biorxiv.org
  26. Cannabidiolic acid, a major cannabinoid in fiber-type cannabis, is an inhibitor of MDA-MB-231 breast cancer cell migration https://www.ncbi.nlm.nih.gov
Disclaimer:
This content is for educational purposes only. The information provided is derived from research gathered from external sources.